Understanding Osteoporosis: Causes and Prevention Information

What Is Osteoporosis and How Does It Develop

Osteoporosis is a medical condition where bones become weak and fragile over time. The name literally means "porous bones." When you have osteoporosis, your bones lose density—meaning they contain less mineral content and become more susceptible to breaking, even from minor falls or, in severe cases, from a simple bump or sneeze.

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To understand osteoporosis, it helps to know how bones work. Bones are living tissue that constantly break down and reform. During childhood and early adulthood, new bone is created faster than old bone is removed, which increases bone mass. Most people reach their peak bone mass around age 30. After that, bone remodeling continues, but more bone is lost than gained.

The development of osteoporosis happens gradually and without symptoms in most cases. You cannot feel your bones becoming weaker. This is why osteoporosis is often called a "silent disease." Many people do not realize they have the condition until they break a bone after a minor fall or injury. Common fracture sites include the hip, spine, and wrist.

According to the National Osteoporosis Foundation, about one in three women over age 70 and one in five men over age 70 will experience a broken bone due to osteoporosis during their lifetime. Approximately 10 million Americans are estimated to have osteoporosis, with another 44 million having low bone mass, which puts them at higher risk.

Bone density is measured using a test called a DXA scan (dual-energy X-ray absorptiometry). This painless test compares your bone density to that of a healthy young adult. Doctors use the results to determine if you have normal bone density, low bone mass (also called osteopenia), or osteoporosis. Understanding where your bone density stands can help guide decisions about prevention and treatment.

Practical Takeaway: Osteoporosis develops slowly over years without obvious signs. Knowing the basics about how bones change helps explain why prevention during younger years and monitoring in later years matters for long-term bone health.

Risk Factors You Cannot Change

Several risk factors for osteoporosis are related to your genetics, age, or sex—things you cannot change. Understanding these unchangeable factors is important because they help determine your overall risk level and whether you might benefit from bone density screening or other preventive measures.

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Age is one of the most significant risk factors. Bone loss accelerates as you get older. For women, the risk increases dramatically after menopause, when estrogen levels drop sharply. Estrogen helps maintain bone density, so the loss of this hormone leads to faster bone loss. Women can lose up to 20% of their bone density in the five to seven years following menopause. Men also lose bone density with age, though typically more gradually than women, and usually starting around age 70.

Sex and gender play a major role in osteoporosis risk. Women are more likely to develop osteoporosis than men. According to the National Institutes of Health, about one in three women over age 50 will have an osteoporotic fracture in their remaining lifetime, compared to about one in five men. This difference is partly due to women's lower peak bone mass and the effects of menopause.

Family history matters significantly. If your parents or siblings have osteoporosis or have experienced fractures from minor injuries, your risk is higher. Genetics account for about 60 to 80 percent of the variation in peak bone mass among individuals. This does not mean you will definitely develop osteoporosis if a family member has it, but it does mean you should be more attentive to prevention strategies.

Ethnicity also influences osteoporosis risk. Non-Hispanic white women and Asian women have higher risk for osteoporosis than Hispanic or Black women. However, this does not mean other groups are without risk—osteoporosis affects people of all ethnicities, and all groups should be aware of their personal risk factors.

Body frame size affects bone density. People with smaller body frames have less bone mass to draw from as they age, which increases fracture risk. Additionally, certain medical conditions present at birth or developing early in life, such as cystic fibrosis or celiac disease, can affect bone development and increase osteoporosis risk.

Practical Takeaway: While you cannot change your age, sex, family history, or ethnicity, knowing whether you have risk factors in these categories helps you make informed decisions about screening, lifestyle changes, and conversations with your doctor about bone health.

Changeable Risk Factors and Lifestyle Choices

The good news is that many risk factors for osteoporosis are within your control. Your daily habits around nutrition, physical activity, and other lifestyle choices significantly influence your bone density throughout your life. Making positive changes now can reduce your fracture risk later.

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Calcium and vitamin D are fundamental to bone health. Calcium is the primary mineral that makes up bone tissue. Vitamin D helps your body absorb calcium and plays a role in bone mineralization. The National Academies of Sciences, Engineering, and Medicine recommends that adults ages 19 to 50 consume 1,000 milligrams of calcium daily, and adults over 50 should consume 1,200 milligrams daily. For vitamin D, adults ages 19 to 70 should get 600 to 800 international units (IU) daily, with slightly higher amounts for those over 70.

Good sources of calcium include dairy products like milk, yogurt, and cheese; leafy green vegetables such as kale, broccoli, and bok choy; fortified plant-based milk alternatives; salmon and sardines with bones; and almonds. If you cannot meet your calcium needs through food, calcium supplements are available, though getting nutrients from food is generally preferred when possible.

Physical activity builds and maintains bone strength. Weight-bearing exercises—where your bones and muscles work against gravity—are particularly beneficial. These include walking, jogging, dancing, hiking, and stair climbing. Muscle-strengthening activities like resistance training, weight lifting, and bodyweight exercises also help. Research shows that people who engage in regular physical activity have higher bone density than sedentary individuals. The U.S. Department of Health and Human Services recommends at least 150 minutes of moderate-intensity aerobic activity per week for adults, combined with muscle-strengthening activities on two or more days per week.

Several lifestyle habits negatively affect bone health. Smoking decreases bone density and increases fracture risk. Alcohol consumption, particularly heavy drinking, can interfere with calcium absorption and damage bone tissue. Excessive caffeine intake may slightly reduce calcium absorption, though moderate caffeine consumption (a few cups of coffee daily) is generally not considered harmful to bone health. Limiting sodium intake is also important, as high sodium consumption can increase calcium loss through urine.

Body weight influences bone density. Being underweight increases osteoporosis risk because there is less weight bearing on bones, and underweight individuals often have lower overall nutrition. However, obesity does not necessarily mean stronger bones—excess weight can sometimes mask low bone density, and obese individuals may still have poor bone quality and fracture risk.

Practical Takeaway: You can take concrete steps today—eating calcium-rich foods, getting vitamin D, exercising regularly, avoiding smoking and excessive alcohol, and maintaining a healthy weight—that directly support bone health and reduce osteoporosis risk over time.

Medical Conditions and Medications That Affect Bones

Beyond age and lifestyle, certain medical conditions and medications can significantly affect bone health. If you have any of these conditions or take these medications, discussing bone health with your healthcare provider becomes even more important.

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Hormonal conditions impact bone density. Hyperthyroidism (overactive thyroid) accelerates bone loss. Conditions affecting the parathyroid glands can disrupt calcium regulation. Diabetes, particularly Type 1 diabetes, increases osteoporosis risk despite sometimes showing higher bone density on scans—the bone quality is compromised even if the quantity appears normal. Cushing's syndrome, caused by excess cortisol in the body, weakens bones. Early menopause or surgical removal of ovaries in women increases risk because of the sudden drop in estrogen.

Digestive system disorders affect calcium and nutrient absorption. Celiac disease damages the small intestine, reducing the ability to absorb nutrients including calcium. Inflammatory bowel diseases like